Kokot F., Hyla-Klekot L., Łoniewski I. Kwasica nieoddechowa – niedoceniony element Kokot F. Zaburzenia gospodarki wodno-elektrolitowej i równowagi. Kokot F, Franek E. Zaburzenia gospodarki wodno-elektrolitowej. In: Zaburzenia gospodarki wodno-elektrolitowej i kwasowo-zasadowej. Kokot F, Franek E (Eds.) . Kokot F., Hyla-Klekot L.: Czujniki sodowe macierzy płynu .. Kokot F., Franek E.: Zaburzenia gospodarki wodno-elektrolitowej i.
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Policy options to reduce population salt intake. Dietary guidelines for Americans. With the notable exception of congenital adrenal hyperplasia CAH and other primary adrenal diseases, in which there is a deficiency in aldosterone synthesis, many other salt-losing disorders share the common feature of inducing secondary hyperaldosteronism.
Nutrition and the prevention of chronic disease: Gospodarka wodno-elektrolitowa i kwasowo-zasadowa w stanach fizjologii i patologii. Short bowel syndrome — surgical perspectives and outcomes. A common complication of chronic renal failure is metabolic acidosis which has a row of negative clinical sequelae including acceleration of the progression of chronic nephropathies.
Sodium chloride and aldosterone: Kokot F, Franek E. The effect of magnesium supplementation on blood pressure: Nerkowa regulacja gospodarki sodowej. Effect of oral tolvaptan in patients hospitalized for worsening heart failure: Metabolic acidosis and kidney disease: Fatal and nonfatal outcomes, kkokot of hypertension and blood pressure changes in relation to urinary sodium excretion.
The key to halting progression of CKD might be in the produce market, not in the pharmacy.
Serum electrolyte patterns in end-stage renal disease. Its societal importance is under-scored by the ever growing number of patients with end-stage kidney disease requiring renal replacement therapy. Charney AN, Donowitz M.
Hyponatremia, hypernatremia and mortality in patients with chronic kidney disease with and without congestive heart failure. Dietary acid reduction with fruits and vegetables or bicarbonate attenuates kidney injury in patients with a moderately reduced glomerular filtration rate due to hypertensive nephronpathy.
Alkalization in patients with metabolic acidosis in the course of chronic kidney disease. Significance of hypo- and hypernatremia in chronic kidney disease. ACE inhibition has adverse renal effects during dietary sodium restriction in proteinuric and healthy rats. Potassium and magnesium — the art of supplementation Abstract Potassium deficiency is the most commonly diagnosed electrolyte disturbance, as it may be caused not only by chronic disease but also by the use of drugs interfering with potassium balance in the kidneys and the digestive tract or shifting potassium in the intra- and extracellular space.
Antenatal excessive sodium intake induces adverse vascular remodelling in offspring. The cardiotonic steroid hormone marinobufagenin induces renal ko,ot, implication of epithelial-to-mesenchymal transition.
Ital J Pediatr ; Turn it on to take full advantage of this site, then refresh the page. Dietary sodium restriction prevents kidney damage in high fructose – fed rats.
Difficulties in diagnosing the cause of hyponatraemia in an extremely premature boy. Paediatr Child Health ; Impact of hospital – associated hyponatremia on selcted outcomes. BMC Pediatr ; Ongoing clinical trials should be able to provide conclusions in the matter of long-term alkalizing treatment and nephroprotection. Consequences and therapy of the metabolic acidosis of chronic kidney disease. Is the sodium ion alone important? Moderation of dietary sodium potentiates the renal and cardiovascular protective effects of angiotensin receptor blocker.
Forum Nefrologiczne ; 5: Unnecessary controversy regarding dietary sodium: Acid-Base Disturbances in Gastrointestinal Disease.
JavaScript is turned off in your web browser. Congenital primary adrenal insufficiency and selective aldosterone defects presenting as salt wasting in infancy: Postgrad Med ; 5: Endokrynol Ped ; 1: Effects of oral sodium bicarbonate in patients with CKD. Jr “Salt-sensitive” essential hypertension in men. Nevertheless, their relevance is limited by the relatively small numbers of subjects and variable stages of chronic kidney disease studied.
Mononuclear phagocyte system depletion blocks interstitial tonicity-responsive enhancer binding protein vascular endothelial growth factor C expression and induces salt-sensitive hypertension in rats. Association of serum bicarbonate levels with mortality in patients with non-dialysis-dependent CKD. Journals Books eBooks Events. Send email Copy url: Franciszek Kokot 1dr hab. N Engl J Med ;